ConclusionsOdontoblastic differentiation was stimulated by SDF?1 activation and repressed by SDF?1/CXCR4 inhibition. Thus, SDF?1/CXCR4 signaling may be a new therapeutic target and strategy to promote repair and regeneration in endodontics.This article is protected by copyright. All rights reserved. (Source: International Endodontic Journal)
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